The US pilots, sprayed in Vietnam military herbicide, threatening dangerous mutations

The Vietnam war ended over 42 years ago, but scientists continue to discover new long term effects of that war for the health of its participants. Researchers from the US found that contact with the herbicide Agent orange may cause precancer.

To combat formations of the army of North Vietnam, the US army, supporting the military conflict 1964-1973, South Vietnam, used the method of defoliation – total destruction of vegetation in the jungle, which was equipped with the base of the North Vietnamese.

Defoliation carried out by spraying from military planes so-called battle of the herbicide Agent orange (Agent Orange), which includes the strongest poison dioxin.

Shortly after the end of hostilities in Vietnam by American military doctors noticed that among the pilots of the U.S. air force sprayed the herbicide, and ground personnel who also had contact with Agent orange increased the incidence of different forms of cancer. In addition, the people exposed to the herbicide, often the children were born with severe developmental defects.

The same phenomenon was observed among the residents of Vietnam, who during the war were exposed to Agent orange while on earth.

Scientists continue to identify previously unknown effects of contact with the herbicide so far. The most recent discovery was the detection among veterans of the US army, exposed to Agent orange, increased risk of development of dangerous disease, is rare among the General population.

We are talking about the violation of hematopoiesis, which is called monoclonal gammopathy of unknown origin. 30% of people with this pathology, which by itself is a benign disease, subsequently developing dangerous oncological disease of the blood and of the blood called multiple myeloma.

Scientists from the cancer Center Sloan-Kettering in new York (Memorial Sloan Kettering Cancer Center in New York City) reported that the pilots and technicians exposed decades ago to Agent orange, compared to the General population the risk of developing monoclonal gammopathy of unknown origin increased 2.4 times.

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